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employed molecular docking and ion resonance biosensors to illustrate the strong binding affinity between PDGFR- and SalB , and SalB relieves HF by inhibiting the PDGFR- signaling pathway and inducing HSC apoptosis, and inflammatory reaction of HSC (Liu et al., 2023b)
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The researchers constructed a model that introduced the ALAS2 missense mutation erythroblasts derived from human cord blood, in which enhanced BACH1 expression was emerged, leading to increased susceptibility to ferroptosis (Ono et al., 2022)
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