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Direct human dose-finding trials are limited

Prior studies have shown that SLC7A11 inhibition, which limits intracellular cysteine availability and reduces glutathione synthesis, increases ROS accumulation, leading to tumor cell vulnerability

The Valproic acid pretreatment regimen opens chromatin, increases the expression of tumor suppressor genes, and enhances the binding of cisplatin to chromatin, ultimately leading to increased cell death (72)

SIRT1-autophagy axis inhibits excess iron-induced ferroptosis of foam cells and subsequently increases IL-1 and IL-18

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Interestingly, upon RNA virus (Sendai virus) and DNA virus (HSV-1) infection, PRMT1 forms oligomers to recruit and methylate the downstream effector TBK1 at R54, R134, and R228, promoting its aggregation and trans-autophosphorylation and the subsequent IRF3-mediated IFN- production [209]