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This connection to autism has been supported by a set of rare primary mitochondrial and nuclear genetic disorders that sometimes have autism as a clinical manifestation (48, 49)

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Activation of endometrial HBP during the window of implantation exerts profound effects on endometrial cell function and embryo implantation

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These distinct mechanisms mean certain patients may respond better to non-GLP-1 options based on their underlying metabolic dysfunction and genetic predispositions

Both approaches achieve similar results