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Within experimental environments, Cagrilintide has been explored for its receptor activity, downstream signaling effects, and stability profile relative to native amylin

The studies began with the determination of the optimal dosing for Cagrilintide in both chow-fed and high-fat diet (HFD)-fed wild-type (WT) mice, followed by analyses of its receptor dependence using receptor activity-modifying protein (RAMP) knockout (KO) models, and mechanistic studies involving neuronal activation and gene expression profiling [2]

Therefore, this review aims to summarize the regulation of copper homeostasis in cardiomyocytes, explore cuproptosis-related targets in cardiovascular diseases, and discuss the translational potential of therapies targeting this pathway

doi: 10.1016/j.vph.2018.02.010