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We observed that Nlrp3 / macrophages present an intrinsically protective redox state and this could be the reason why macrophages did not produce ROS after sUA+LPS stimulus

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HMGB1, ATP, and uric acid further stimulate macrophages to secrete IL-1 and induce pulmonary epithelial cells to release the chemokine CCL2, processes that collectively influence the fibrotic progressio [247, 314] (Fig