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Hi Nackapan, Yes, I would agree with a lot of what you say

Implications of CT2 in Cancer Unlike the restricted expression in healthy tissue, CT2 is largely expressed in several cancer types also originating from tissues that normally do not express this protein (Lal et al., 2007

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When taking into account recurrent episodes and seasonal variation, NTHi accounts for approximately 60% of cases (Haggard, 2008

In the case of epilepsy, impaired K + buffering, glutamate clearance, lipid accumulation and malfunctioning of the glutamate (GABA)-glutamine shuttle emerge as leading pathophysiological processes

In general, by either excess or impairment, NO appears to cause or contribute to a variety of disturbances, in particular, hypertension, angina, impotence, circulatory shock, septic shock, stroke, inflammation, respiratory distress syndromes, pulmonary hypertension, adhesion and aggregation of platelets and leukocytes, diabetes, hypotension, and Parkinson's disease