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The affinity profile is calibrated for triple balance: full agonist at GIPR, full agonist at GLP-1R, partial agonist at GCGR, preserving glycemia while exploiting glucagon thermogenic benefits

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Central GLP-1 neurones in the brainstem react to stomach distension and meal consumption, conveying satiety signals

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I held onto that mental model longer than I should have as a student, and it made everything about bone metabolism harder to understand than it needed to be