This fragment-based approach is what differentiates AOD-9604 from GHRH analogs such as Tesamorelin, which act upstream in the pituitary rather than directly on metabolic pathways
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The pro-inflammatory cytokines are excessively secreted from WAT in either genetic-based or diet-induced obese mice, which in turn interrupt insulin action in adipocytes, contributing to the onset and development of insulin resistance [60]
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