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Diabetes 64(7):23982408 Gao Q et al (2012) Clostridium butyricum activates TLR2-mediated MyD88-independent signaling pathway in HT-29 cells

The clinic conducts regular follow-ups to assess for any developing complications

Tesaglitazar is a PPAR/ dual-agonist, which in phase II and III clinical trials improved glucose and lipid metabolism with greater efficacy relative to selective PPAR agonism 13,17,18,19,20,21