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IGF-1 LR3 is engineered to reduce binding to inhibitory IGF-binding proteins, thereby increasing its interaction with IGF-1 receptors in target tissues like skeletal muscle. Clemmons, D

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(3) Targeting Vasocclusion Increased expression and activation of normally inactive erythroid adhesion molecules promote cytoadherence of sickle RBCs to the endothelium accompanied by platelets and leukocytes