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Surprisingly, mice with specific genetic deletion of Gpx1 appear phenotypically normal and with normal life span ( 2 O 2 scavenging in Gpx1 deficiency

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(2) The Microsomal Ethanol Oxidizing System (MEOS): This pathway involves the metabolism of ethanol to acetaldehyde in the mitochondria, converting NADPH and O 2 into oxidized nicotinamide adenine dinucleotide phosphate (NADP+) and H 2 O, while simultaneously producing ROS (Lu and Cederbaum, 2008)