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These different signaling pathways, which are associated with ER activation and ER inhibition, respectively, depend on cell context with the balance of receptor activation determining the overall response of the cell 78

Activation of these pathways would increase intracellular ROS levels by inhibiting the transcription factor forkhead box transcription factor O (FOXO), which boost the production of antioxidants such as SOD2, catalase, and sestrin3 [28]

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Depending on the injury, metabolic failure also occurs, leading to mitochondrial dysfunction (i.e., reduced respiratory rates and production of adenosine 5-triphosphate [ATP] and calcium overload) (see also Sullivan in Appendix C)