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The following era (1985 to present) of molecular genetics, recombinant technology and the generation of genetically modified biological systems has expanded our understanding of the regulation and role of the GHIGF axis

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Immune dysregulationparticularly involving T-helper cell imbalance, increased pro-inflammatory cytokines (e.g., TNF-, IL-6), and reduced regulatory T cell functionhas been shown to impair maternal-fetal tolerance

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