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In studies, BPC 157 has demonstrated its ability to counteract the harmful effects of neurotoxins, reducing cell damage and supporting neuronal health

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The pathophysiology involves energy failure during ischemia and oxidative stress during reperfusion, with local acidosis exacerbating cellular damage through anaerobic glycolysis [4,5,6]

Quantification of GSH in BAL fluid was performed as reported by Steele et al