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It has been proposed that direct screening for these pathogenic variants would be appropriate in these populations.[58-61] A founder pathogenic variant at the 3 splice acceptor site of intron 3 in the XPA gene is present in approximately 1% of the population in Japan representing nearly 1 million people.[62] The function of the XP genes is to recognize and repair photoproducts from UV radiation

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The neuroprotective effects of GLP-1R agonists may be closely related to their impact on insulin resistance and glucose metabolism

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