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(2001, Int J Obes) showed that chronic treatment with AOD-9604 restored repressed beta-3 AR mRNA levels in obese mice toward lean-mouse values, characterizing a chronic-treatment mechanism that complements the fragment's acute lipolytic action

These results suggest that blocking AMPK activation to disable energy sensing of cancer cells may functionally abrogate tumor growth under energy stress but there is currently a lack of actionable therapeutic strategies to this end

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A defective cell migration phenotype is thus consistent with decreased metastasis/increased survival of lung cancer patients with high MS4A15 -expressing tumors (Supplementary Fig

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Despite the benefits of anticoagulant treatment with dabigatran, liver injury was significantly aggravated 24 h after APAP administration due to a decrease in hepatocyte proliferation