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J Vasc Bras

More than thirty years ago, Coleman and Blass[22] suggested an abnormality in carbohydrate metabolism in individuals with ASD, several years later, it was proposed that ASD may be a disorder associated with an impairment in mitochondrial function[23]

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Most notably, HO-1 expression was significantly decreased in the FAC-treated group after HO-1 knockdown, with a continued decreasing trend after theaflavins treatment (Fig