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Clin Biochem (2013) 46:30812

IGF-1 LR3 differs from endogenous IGF-1 in one critical way: the arginine substitution at position 3 prevents binding to IGF-binding proteins (IGFBPs), extending circulation time and allowing the peptide to remain bioavailable far longer than native IGF-1

The exact molecular mechanisms leading to WL and glycaemia improvements with tirzepatide are still under investigation, mainly because the role of GIP receptor activation with tirzepatide is unclear

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