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Description

Mitochondrial dysfunction as a mechanism of drug-induced hepatotoxicity: Current understanding and future perspectives

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doi: 10.3389/fendo.2023.1268308

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1998) wykazywa profil selektywny : pobudza uwalnianie GH przez receptor greliny GHS-R1a bez istotnego wzrostu kortyzolu i prolaktyny, z ograniczonym wpywem na ACTH

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