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Similarly, exenatide enhanced the efficacy of programmed cell death protein-1 (PD-1) blockade in LLC- and MC38-based lung and colon cancer models (109), and liraglutide did so in LLC- and Hepa1-6-bearing mice, mainly by attenuating neutrophil extracellular trap (NET)- and ROS-driven immunosuppression and thereby permitting stronger CD8+ T-cell effector responses (110)

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