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However, other studies suggest that iron overload does induce ferroptosis in endometriotic stromal cells or specific subpopulations (like mesenchymal stem cells), and that this process, rather than eliminating cells, might paradoxically promote inflammation and fibrosis [94]

M2 macrophages are anti-inflammatory, promoting tissue repair and resolution of inflammation

Overall, increasing NAD + levels stimulated mitochondrial function, promoting mitophagy and preventing cellular senescence

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Another study supports the idea of interplay between apoptotic cell identity, metabolic status, and phagocyte receptors in determining the degradation pathway and the immunological outcomes of efferocytosis