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Elucidating the contribution of mitochondrial glutathione to ferroptosis in cardiomyocytes

GlyNAC (Glycine and N-Acetylcysteine) Supplementation in mice increases length of life by correcting glutathione deficiency, oxidative stress, mitochondrial dysfunction, abnormalities in mitophagy and nutrient sensing, and genomic damage

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doi: 10.1002/hsr2.1640 52 ChandrasekaranPWeiskirchen SRW

Plaque ruptures and clog the blood vessels leading to lesser blood flow to heart resulting in heart stroke