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By blocking NF-B, it reduces the production of pro-inflammatory cytokines (IL-1, TNF-, IL-6) and inflammatory mediators (PGE2, NO) in keratinocytes and dermal cells

doi: 10.1093/jnci/djm117

DengYWangMTianTLinSXuPZhouLet al

Laboratory findings help explain why scientists remain interested, but they cannot establish clinical benefit by themselves

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