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Recent research has indicated that wild-type p53 can promote ferroptosis in certain contexts (e.g., by repressing SLC7A11), whereas selected gain-of-function mutants may also sensitize tumor cells to ferroptosis, although many mutants confer resistance (70)
The second function of the chylomicron is to transport cholesterol to the liver, although on the way the cholesterol may be taken up by tissues including the atheromatous plaque where the macrophage sits in waiting with a specific apo B48 receptor [36] as well as VLDL and scavenger receptors [35]
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