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4.4 Neonatal hypoxicischemic encephalopathy The pathophysiology of HIE involves hypoxicischemic injury, characterized by perturbations in mitochondrial dynamics and the inhibition of critical energy-producing pathways, including oxidative phosphorylation and fatty acid -oxidation ( -oxidation and its prior neuroprotective effects in adult ischemia models, it represents a biologically plausible candidate for investigation in neonatal brain injury

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This lack of mention is significant, as pharmaceutical companies are required to report all observed and statistically relevant adverse events during clinical trials

In every stage of disease walking improved: Stage 3: (96%), Stage 2: (88%), Stage 1:(33%)

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