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This culminates in immune destruction of the epithelium in exocrine glands, which causes the most common manifestations of the disease

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This accumulation is mediated by plasmalemmal transporters predominantly expressed in glial cell, such as divalent metal transporter 1 DMT1/SLC11A2 or zinc transporter ZIP14/SLC39A14, 374,375 which both transport Fe 2+ (ferrous), whereas Fe 3+ (ferric) is accumulated through transferrin receptors

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There is further epigenetic evidence that epithalon can regulate neuron differentiation gene expression [5]