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In addition to supplementation, it may also be necessary to make dietary changes to ensure adequate intake of vitamin B12-rich foods
Consequently, beneficial effects can be directly related to the reversal of obstructive injury outcome consequences, in both early (IC rats
The proposed mechanism involves controlling glutamate release in the synaptic cleft, modulating NMDA receptor stimulation, affecting excitatory amino acid transporter (EAAT)-mediated reuptake, and maintaining the glutamate/glutamine cycle within astrocytes
Individuals who are low in B12 may experience persistent fatigue, weakness, or lightheadedness, along with difficulties concentrating or a sense of brain fog
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