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Figure 2C highlights the immunological arm of this axis, showing how SCFAs and related metabolites polarize macrophages toward an M2-like phenotype, promote Treg differentiation and increase IL-10/TGF- production, thereby dampening systemic and islet inflammation and indirectly preserving -cell function

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Combined with our trafficking and insulin release results, these data show a canonical role for -arrestin recruitment in promoting GLP-1R endocytosis and desensitization

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The healthy development of oocytes and subsequent fertilization depend on these cells, which multiply before ovulation [213]