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Originally developed by researchers at Washington State University, it was designed to explore the cognitive role of the AT4 receptor

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Intrathecal administration of exenatide upregulated IL-10 and -endorphin expression in spinal microglia of neuropathic rats [63]

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These animals produced low systemic levels of NO at 12 h of infection compared to saline-CLP group, increasing this production at 24 h post infection (Figure 2H)