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Enhanced cardiac akt/protein kinase B signaling contributes to pathological cardiac hypertrophy in part by impairing mitochondrial function via transcriptional repression of mitochondrion-targeted nuclear genes

Long-chain fatty acyl-CoA thioesters cannot enter the mitochondrial matrix in animals because they are not able to pass through the inner mitochondrial membrane

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Low glutathione compromises liver function, which works to flush the body of damaging free radicals

Methods 44 , 200204

10.1016/j.foodchem.2011.06.047 85 RajeshA