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(2008) demonstrated that systemic administration of IGF-1 LR3 to dystrophic mdx mice (a model of Duchenne muscular dystrophy) significantly reduced susceptibility to contraction-mediated damage in extensor digitorum longus, soleus, and diaphragm muscles

Conversely, unresponsive patients show minimal changes in outcomes, suggesting that factors beyond L- carnitine adequacy may contribute to their unresponsiveness

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