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doi: 10.1161/strokeaha.109.569830 143 PalombellaS.GuiottoM.HigginsG

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The homeostatic mechanism of cellular respiration is known to be regulated by two stress-sensing protein kinases i.e., target of rapamycin (TOR) (Ma and Blenis 2009) and AMP-activated protein kinase (AMPK) (Hardie and Carling 1997), often playing an inhibitory role

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