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Targeting Wnt/-catenin and BCL-2, Two Critical Survival Factors, Synergistically Induces Apoptosis in AML via Rac1-mediated MCL-1 inhibition

Data availability The proteomic datasets generated during this study have been deposited to the ProteomeXchange Consortium ( via the PRIDE repository (identifier: PXD056759)

Epidemiological research suggests that ASD arises from complicated interactions between genetics and environment, but the precise mechanism by which this occurs remains unclear (Hallmayer et al., 2014)

336 Progressive loading and exercise influence TGF- signaling pathways, affecting ECM composition and the repair process

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Additionally, lipotripeptides (DICAMs) inhibit and disrupt bacterial biofilms, and GPE supports neuroprotection and regeneration through ERK and PI3K/Akt signaling activation