Acknowledgments The author is grateful to the communities of structural biology, biophysics, medicinal and computational chemistry and algorithm design, for the continued accumulation of knowledge and data for drug discovery & design, and for the continued development of tools (hardware, software and algorithm) for drug discovery & design.
We can conclude that continuous endoglin overexpression produces unstable vessels with poor mural coverage, which could contribute to increase the vascular permeability and the breakage of the new vessels
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