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The mechanism behind this enhancement was attributed to the increased availability of active binding sites introduced by the amine functional groups, which form strong coordination bonds with metal ions, improving their retention on the adsorbent surface

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Evidently, intracellular cyst(e)ine contribute to ferroptosis suppression via at least two additional pathways in different contexts: in a mouse lung adenocarcinoma model, NFS1 cysteine desulfurase utilizes cysteine to synthesize ironsulfur clusters, which are necessary for preventing activation of the iron-starvation response, subsequent iron influx and ferroptosis 14 (Fig

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