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This is caused by an imbalance between factors that relax the endothelium, such as nitric oxide (NO), prostacyclin (PGI 2 ), and down regulation of endothelioum-dependent hyperpolarization (EDH) and factors that cause contraction such as thromboxane A 2 (TxA 2 ) and upregulation of endothelin-1 (ET-1)

Moreover, the other inflammatory cytokines like adhesion molecules would result in vascular dysfunction and consequently neuropathic pain (13)

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With the further understanding of the unique energy metabolism feature of mitochondria in a distinct population of immunocytes, tissue or organ-specific therapeutics of mitochondria-targeting intervention may provide promising strategies for SLE treatment

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