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These results thus show that spike protein-dependent loss of CFTR, and rescue of lost CFTR by cardiac glycoside drugs, can be detected in epithelial cells other than lung, and in cells for which CFTR is recombinant rather than native
In laboratory research, it is used to investigate fibroblast activation, collagen and elastin synthesis, wound repair cascades, oxidative stress modulation, and gene expression related to tissue remodeling and cellular aging
10.4103/1319-3767.37808 24 BenoyI
[DOI] [PMC free article] [PubMed] [Google Scholar] Ahmed S, Anuntiyo J, Malemud CJ, Haqqi TM
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