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Reversely, AMPK-mediated phosphorylation of Beclin-1 facilitates ferroptosis by directly inhibiting the activity of cys/glu antiporter system Xc-

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Preventing this decrease, with the administration of a SIRT-1 activator, reversed ROS bursts, increased downstream nuclear PGC-1a and Nrf2 expression, and attenuated increases in mitochondrial membrane potential and neuronal apoptosis induced by ischemia/reperfusion injury [78]

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