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Abstract The enzyme-catalyzed oxidation of tyrosine in living organisms leads to the formation of dopachrome which is a key intermediate in the melanogenesis pathway

Growth hormone/IGF-1 axis supports bone and cartilage metabolism (established endocrinology)

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It is unclear why inhibition of c-MET by foretinib increased radiosensitivity, whereas inhibition with crizotinib appeared to have the opposite effect, though this phenomenon is likely due to other kinases targeted by these two molecules