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Only the administration of insulin directly into the portal vein will really treat the metabolic derangement present in T1DM (Fig

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Hence, prolonged IGF-1 or IGF-1 gene therapy could disrupt the nuclear p53progerin interaction to repress oxidative SIPS via activating the PI3K/AKT1 pathway and inhibiting p53 nuclear translocation

However, the only statistically significant decrease was seen in the chlorophyllin-contained groups in tissue