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The anorectic effects of GLP-1 agonism are reported to result from the suppression of AgRP/NPY neurons and activation of POMC/CART neurons, and these responses occur upstream of MC4R [28, 29]

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In our study, the requirement of endogenous GLP-1R signaling to increase small intestinal length was only observed cold-stressed male, but not female mice, suggesting a sex difference in the identity or action of compensatory intestinotrophic signaling molecules