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Constitutive activation of NRF2, mainly via loss-of-function mutations in the CUL3-adapter KEAP1 or gain-of-function mutations in NFE2L2 , is thought to provide a fitness advantage to tumor cells in confronting metabolic/oxidative stress resulting from dysregulated growth (DeNicola et al, 2011

It adds a third receptor, glucagon, on top of GIP and GLP-1

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Although the mechanism for maintaining sodium/fluid balance is separate from satiety regulation, patients can have an increased risk for dehydration while on GLP-1 medications, Dr

it does not describe clinical use or outcomes

For full prescribing details, refer to the electronic Medicines Compendium (eMC) Summary of Product Characteristics (SmPC) for each agent