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This inhibition preserves cellular NAD+ levels by reducing their consumption, simultaneously increasing GLUT4 transporter expression and activating sirtuin 1, known as the "longevity gene." The compound exhibits high membrane permeability and selectivity without affecting related methyltransferases or NAD+ salvage pathway enzymes, creating targeted metabolic effects while maintaining cellular safety

Immunity 56 , 17941808.e8 (2023)

Overall the above reported evidence on the involvement of GSH and the redox environment are only some issues that deserve to be further investigated to comprehend the several aspects characterizing the possible crosstalk between redox reactions and modulation of ATG

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