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Apr 16 2020;9(4)doi:10.3390/cells9040990

Authors propose the following model of PLC-mediated intestine cancer development: stem cells first undergo neoplastic transformation and begin to express more PLC, and then this leads to the expression of angiogenic factors, which drives further tumor development

Therapeutically, this raises two complementary possibilities: (1) SQOR-high tumors may exhibit enhanced mitochondrial ferroptosis resistance through maintained CoQ 10 H 2 and sulfur-based RTAs and (2) tumors with altered sulfur/selenium metabolism may reveal synthetic vulnerabilities when SQOR-linked defense is impaired

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Are these drugs still promising, or was the hype misplaced

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