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In the mitochondria, LC is converted to ALC, and balances the acetyl CoA/CoA ratio to maintain glucose metabolism through the TCA cycle, yielding a higher energy production [34]

From a clinical lens, determining the optimal ways to modulate the NRF2 pathway in stem cells, ex vivo for cell transplantation, or in directly in vivo through the development of effective small molecule inhibitors acting upstream or downstream of NRF2, will be critical

Mitochondria are little organelles (organs of the cell, so to speak) that break down nutrients and create energy

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This matters because higher systemic glutathione levels have a greater capacity to modulate GST enzyme activity and, theoretically, alter the conjugation of other hepatically processed drugs [6]

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