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In vitro, RBL-2H3 cells pre-treated with luteolin (10 M) showed inhibition of IL-4 expression and mast cell degranulation[164]

The honest caveat: long-term human safety data does not exist

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an HMGCR-independent pathway, including allosteric hindrance of lymphocyte function-associated antigen 1 (LFA-1), which represses activation, proliferation, and cellular migration of T cells [40] and HMGCR-dependent mechanisms, which rely on their ability to prevent prenylation of small GTPases [41, 42]

This leads to improved nerve function and helps prevent long-term nerve complications, offering both short- and long-term benefits for patients with neuropathies

In 1998, Duport et al