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Researchers continue to investigate AOD-9604 peptide in laboratory studies involving peptide chemistry, molecular signalling, receptor interactions, and experimental biochemistry under controlled research conditions

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found that these two vectors were highly expressed in myocardial mitochondria and showed that inhibition of DIC and OGC increased mitochondrial ROS and GSH depletion, exacerbating ferroptosis in the myocardium [60]

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Mutations in ELAC2 associated with hypertrophic cardiomyopathy impair mitochondrial tRNA 3-end processing