The table below summarizes the core methodological features and provides a quality rating for each study, helping to contextualize their contribution to clinical recommendations
6o), the contributions to the maximal OCR value in each genotype by ATP-linked OCR , proton leakage, reserve capacity, and non-mitochondrial OCR were altered by Gclc deficiency (Fig
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Attenuation of doxorubicin-induced cardiotoxicity by mdivi-1: a mitochondrial division/mitophagy inhibitor
GLP-1 Receptor Glucagon-like peptide-1 receptor