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The Src family kinases Hck and Fgr are dispensable for inside-out, chemoattractant-induced signaling regulating 2 integrin affinity and valency in neutrophils, but are required for 2 integrin-mediated outside-in signaling involved in sustained adhesion

Structure of a complete integrin ectodomain in a physiologic resting state and activation and deactivation by applied forces

[1] These changes make MT-1 less susceptible to enzymatic degradation

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Angiographic evaluation of coronary microvascular dysfunction in patients with heart failure and preserved ejection fraction