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Several clinical studies have demonstrated that patients receiving vitamin E, selenium, and/or N-acetylcysteine (NAC, a glutathione precursor) as adjunctive treatments alongside standard M

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However, at later time points, we observed diminished steady state expression of NHK in etomoxir-treated cells, and this distinction was lost when degradation of NHK was blocked by the proteasomal inhibitor MG-132 (Figure 3D)